Increased expression of soluble form of vascular cell adhesion molecule-1 aggravates autoimmune arthritis in MRL-Fas^<Ipr> mice

• OISHI Hisashi

  Department of Anatomy and Embryology, Institute of Basic Medical Sciences, Graduate School of Comprehensive Human Sciences, Biomolecular and Integrated Medical Sciences, University of Tsukuba

• MIZUKI Shinichi

  Department of Pathology, Division of Pathogenomics, Ehime University Graduate School of Medicine

• TERADA Miho

  Department of Pathology, Division of Pathogenomics, Ehime University Graduate School of Medicine

• KUDO Megumi

  Department of Pathology, Division of Pathogenomics, Ehime University Graduate School of Medicine

• ARAKI Kimi

  Institute of Molecular Embryology and Genetics, Kumamoto University

• ARAKI Masatake

  Institute of Resource Development and Analysis, Kumamoto University

• NOSE Masato

  Department of Pathology, Division of Pathogenomics, Ehime University Graduate School of Medicine

• TAKAHASHI Satoru

  Department of Anatomy and Embryology, Institute of Basic Medical Sciences, Graduate School of Comprehensive Human Sciences, Biomolecular and Integrated Medical Sciences, University of Tsukuba

Journal

• Pathology international

  Pathology international 57 (11), 734-740, 2007-11-01

Keywords

• autoimmunity
• CD106
• mouse model
• MRL mouse
• rheumatoid arthritis
• rheumatoid factor
• vascular cell adhesion molecule-1

Details

• CRID: 1572261550501238656
• NII Article ID: 10021234200
• NII Book ID: AA10984364
• ISSN: 13205463
• Web Site: https://search.jamas.or.jp/link/ui/2008142042
• Text Lang: en
• Data Source:
  • CiNii Articles