Hyperkalemia PRobably Reverses the Antiarrhythmic Effects of Amiodarone -A Case Report-

• AKIYAMA Jun-ichi

  Department of Cardiology,Kasukabe Shuwa Hospital

• TOMIZAWA Tamami

  Department of Cardiology,Kasukabe Shuwa Hospital

• UMEZAWA Shigeo

  Department of Cardiology,Kasukabe Shuwa Hospital

• MORISHITA Akira

  Department of Cardiology,Kasukabe Shuwa Hospital

抄録

Sustained monomorphic ventricular tachycardia (VT) developed in a 58-year-old man with acute myocardial infarction and end-stage renal disease. Amiodarone was effective in preventing VT recurrence. Sustained VT was not induced during an electrophysiologic study. However, VT recurred during accidental hyperkalemia, which was caused by the change of dialysis therapy from peritoneal dialysis to hemodialysis. VT subsided with correction of hyperkalemia. Thereafter, VT did not recur as long as the serum potassium concentration was kept within the normal range. Several months later, the patient died suddenly because poor dietary compliance resulted in an increase in his potassium concentration. This case suggests that hyperkalemia may reverse the potent antiarrhythmic effects of amiodarone.

収録刊行物

• Japanese circulation journal

  Japanese circulation journal 63 (4), 323-325, 1999-03-20

  社団法人日本循環器学会

キーワード

• Amiodarone
• Myocardial infarction
• Renal failure
• Ventricular tachycardia

詳細情報

• CRID: 1573387451907534080
• NII論文ID: 110002572839
• NII書誌ID: AA00690731
• ISSN: 00471828
• Web Site: https://search.jamas.or.jp/link/ui/1999210391
• 本文言語コード: en
• データソース種別:
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