SP-D as a Marker of Amiodarone-induced Pulmonary Toxicity.

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  • UMETANI Ken
    The Second Department of Internal Medicine, Yamanashi Medical University
  • ABE Makoto
    The Second Department of Internal Medicine, Yamanashi Medical University
  • KAWABATA Kenichi
    The Second Department of Internal Medicine, Yamanashi Medical University
  • IIDA Takashi
    The Second Department of Internal Medicine, Yamanashi Medical University
  • KOHNO Isao
    The Second Department of Internal Medicine, Yamanashi Medical University
  • SAWANOBORI Takao
    The Second Department of Internal Medicine, Yamanashi Medical University
  • KUGIYAMA Kiyotaka
    The Second Department of Internal Medicine, Yamanashi Medical University

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Two patients with amiodarone-induced pulmonary toxicity (APT) showed abnormally increased serum SP-D concentrations, although their KL-6 level was within the normal range. In a 59-year-old man with ischemic heart disease, APT progressed rapidly and required steroid pulse therapy. During the clinical course, SP-D was as high as 375 ng/ml, although the KL-6 level was only 289 U/ml. In a 58-year-old man treated for dilated cardiomyopathy, SP-D increased to 289 ng/ml, while KL-6 remained at less than 500 U/ml. These cases indicate that SP-D is a useful and early diagnostic marker for APT even when KL-6 is not elevated.<br>(Intrenal Medicine 41: 709-712, 2002)

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  • Internal Medicine

    Internal Medicine 41 (9), 709-712, 2002

    一般社団法人 日本内科学会

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