Ataxic Hemiparesis Following Thalamic Lacunar Infarction.

DOI Web Site PubMed 被引用文献1件
  • EMORI Tetsuro
    the Cerebrovascular Division, Department of Medicine, National Cardiovascular Center
  • KURIYAMA Yoshihiro
    the Cerebrovascular Division, Department of Medicine, National Cardiovascular Center
  • IMAKITA Satoshi
    Department of Radiology, National Cardiovascular Center
  • SAWADA Tohru
    the Cerebrovascular Division, Department of Medicine, National Cardiovascular Center

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A 60-year-old man developed left hemiparesis and homolateral ataxia with normal sensation and normal somatosensory evoked potentials. A lacunar infarct with gadolinium enhancement in the right dorsolateral part of the thalamus was demonstrated on magnetic resonance imaging. Thalamic lesion is a relatively rare cause of ataxic hemiparesis ; most of the reported cases of ataxic hemiparesis caused by thalamic lesion were accompanied by sensory disturbances. This is an interesting case which suggested that the thalamic lesion could be responsible for the ataxic hemiparesis without a sensory disturbance.<br>(Internal Medicine 31 : 889-892, 1992)

収録刊行物

  • Internal Medicine

    Internal Medicine 31 (7), 889-892, 1992

    一般社団法人 日本内科学会

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