Dolichoectatic Vertebrobasilar Dissecting Aneurysm Originating from Atherosclerosis: An Autopsy Case
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- Isa Katsunori
- Department of Cardiovascular Medicine, Nephrology, and Neurology, University of Ryukyus Graduate School of Medicine, Japan
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- Sakima Hirokuni
- Department of Cardiovascular Medicine, Nephrology, and Neurology, University of Ryukyus Graduate School of Medicine, Japan
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- Kosuge Noritake
- Department of Pathology and Oncology, University of Ryukyus Graduate School of Medicine, Japan
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- Kokuba Kazuhito
- Department of Cardiovascular Medicine, Nephrology, and Neurology, University of Ryukyus Graduate School of Medicine, Japan
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- Goya Yoshino
- Department of Cardiovascular Medicine, Nephrology, and Neurology, University of Ryukyus Graduate School of Medicine, Japan
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- Nakachi Koh
- Department of Cardiovascular Medicine, Nephrology, and Neurology, University of Ryukyus Graduate School of Medicine, Japan
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- Ishihara Satoshi
- Department of Cardiovascular Medicine, Nephrology, and Neurology, University of Ryukyus Graduate School of Medicine, Japan
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- Tokashiki Takashi
- Department of Cardiovascular Medicine, Nephrology, and Neurology, University of Ryukyus Graduate School of Medicine, Japan
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- Ohya Yusuke
- Department of Cardiovascular Medicine, Nephrology, and Neurology, University of Ryukyus Graduate School of Medicine, Japan
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- Saio Masanao
- Department of Pathology and Oncology, University of Ryukyus Graduate School of Medicine, Japan
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抄録
We herein report an unusual case of profound brain infarction of the posterior circulation due to a dolichoectatic vertebrobasilar dissecting aneurysm (DVDA) originating from atherosclerosis. On autopsy, diffuse atherosclerosis was observed with a multi-fusiform aneurysm measuring 1 to 2 cm in diameter ranging from the left vertebral artery to the basilar artery. The microscopic findings of the aneurysm revealed severe stenosis of the artery caused by intimal thickening, intimal flap formation and thrombosis, indicating the presence of a dissecting aneurysm originating from atherosclerosis. The DVDA observed in this case was considered to be slowly progressive and associated with the development of atherosclerosis. The etiology of structural destabilization in patients with DVDA involves rupture of the internal elastic lamina, which is dislodged by massive hematomas that form atheromatous lesions.<br>
収録刊行物
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- Internal Medicine
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Internal Medicine 52 (16), 1821-1823, 2013
一般社団法人 日本内科学会
