Electrophysiological Studies in Slowly Induced Sea and Fresh Water Drowning in Rabbits

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  • FUJIMURA KAZUAKI
    Department of Legal Medicine, Yamaguchi University School of Medicine
  • KOMURA SETSUO
    Department of Forensic Medicine, Shiga University of Medicine Science
  • FURUNO JUNJI
    Department of Legal Medicine, Yamaguchi University School of Medicine
  • YAMAMOTO YOSHIO
    Department of Forensic Medicine, Shiga University of Medicine Science
  • MATSUDA NAONOBU
    Department of Forensic Medicine, Shiga University of Medicine Science
  • YOSHIMOTO KANJI
    Department of Forensic Medicine, Shiga University of Medicine Science

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  • Electrophysiological Studies on Slowly Induced Sea and Fresh Water Drowning in Rabbits
  • Electrophysiological Studies in Slowly

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Surface and deep electroencephalograms, electrocardiogram and behavior of 10 male rabbits were studied by infusing fresh water or sea water slowly at a speed of 0.5-2.0ml/min, by means of a dripping apparatus, into the canulated trachea until the death of the animals. From the behavior of the animal and EEG and EGG findings, the course was divided into 4 stages. The let, alert stage; 2nd, adapted or calm stage; 3rd, dyspnea stage; and 4th, covering from the agonal spasm to ECG silence. The course of this drowning death was quite different from that due to a large amount of water aspirated within a short period. The volume of fresh water needed to kill the animals was 7 times larger than that of the sea water in this method, and the latter was less than half of the volume of sea water necessary for the death due to short period aspiration. The mechanism of death seems to be slow and prolonged asphyxia and, among the terminal events in fresh water drowning, there might occur marked pulmonary hypertension that results in a high degree of lung edema. In the ECG at terminal stage, final bradycardia (84.4% of normal pulse rate) at around the time of surface EEG disappearance, and final tachycardia (142.7% of normal pulse rate) about 120 sec later than final bradycardia and 40 min later than deep EEG disappearance were observed consistently. After clinical signs of death ECG still continued to beat slowly for 1-2 hr.

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