Hypothyroidism Enhanced Portal Hypertension in a Patient with Alcoholic Liver Cirrhosis, Resulting in the Development of Ascites
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- Kakisaka Keisuke
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Endo Kei
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Suzuki Akiko
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Hayashi Sadanari
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Abe Tamami
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Yoshida Yuichi
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Oikawa Takayoshi
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Miyamoto Yasuhiro
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Sawara Kei
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Ishida Kinji
- The Center for Electron Microscopy and Bio-Imaging Research, Iwate Medical University, Japan
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- Kuroda Hidekatsu
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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- Takikawa Yasuhiro
- Division of Hepatology, Department of Internal Medicine, Iwate Medical University, Japan
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抄録
A man diagnosed with alcoholic liver cirrhosis complained of abdominal distention due to massive ascites. The ascites did not resolve with diuretic agents. The serum-ascites albumin gradient value of 1.9 g/dL and the total protein level in the ascites of 3.1 g/dL indicated the ascites to have been caused by portal hypertension. Hypothyroidism was detected, and the patient received supplementation with levothyroxine. The ascites dramatically decreased after supplementation with levothyroxine. We herein conclude that the ascites in the present case had thus been strongly influenced by portal hypertension, which was induced by liver dysfunction associated with liver cirrhosis and hypothyroidism.<br>
収録刊行物
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- Internal Medicine
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Internal Medicine 54 (18), 2327-2331, 2015
一般社団法人 日本内科学会