Gene Mutations Responsible for Overexpression of AmpC β-Lactamase in Some Clinical Isolates of<i>Enterobacter cloacae</i>

DOI Web Site 被引用文献4件
  • Kenichi Kaneko
    Department of Environmental Infectious Diseases, Graduate School of Medical Sciences
  • Ryoichi Okamoto
    Department of Environmental Infectious Diseases, Graduate School of Medical Sciences
  • Ryuichi Nakano
    Department of Environmental Infectious Diseases, Graduate School of Medical Sciences
  • Sayoko Kawakami
    Department of Central Laboratory, School of Medicine, Teikyo University, 2-11-1, Kaga, Itabashi-ku, Tokyo, Japan
  • Matsuhisa Inoue
    Department of Environmental Infectious Diseases, Graduate School of Medical Sciences

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<jats:title>ABSTRACT</jats:title><jats:p>AmpC regulatory genes in 21 ceftazidime-resistant clinical isolates of<jats:italic>Enterobacter cloacae</jats:italic>(MICs of ≥16 μg/ml) were characterized. All isolates exhibited AmpC overproduction due to AmpD mutation. Additionally, we found two AmpR mutants among the isolates. This is the first report of chromosomal<jats:italic>ampR</jats:italic>mutation in clinical isolates of<jats:italic>E. cloacae</jats:italic>.</jats:p>

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