Induction of Tumor Necrosis Factor-.ALPHA. mRNA in the Kidney of the Mouse Chronic Hepatitis Model.

• Okamoto Toshihiro

  Research Laboratories, Nippon Chemiphar Co., Ltd.

• Furuya Masami

  Research Laboratories, Nippon Chemiphar Co., Ltd.

• Yamakawa Tomio

  Research Laboratories, Nippon Chemiphar Co., Ltd.

• Yamamura Ken-ichi

  Institute of Molecular Embryology and Genetics, Kumamoto University School of Medicine

• Hino Okio

  Department of Experimental Pathology, Cancer Institute

Bibliographic Information

Other Title

• Induction of Tumor Necrosis Factor-α mRNA in the Kidney of the Mouse Chronic Hepatitis Model
• Induction of Tumor Necrosis Factor アルファ

Abstract

Previously we reported the induction of tumor necrosis factor (TNF)-α mRNA in the liver of interferon (IFN)-γ transgenic mouse, which might be the result of IFN-γ gene expression. In the present study, IFN-γ mRNA was induced in the liver, kidney and lung. TNF-α mRNA was, however, induced in the liver and kidney, but not in the lung. Induction of interleukin (IL)-2 and IL-1β mRNA was lacking in any of these organs. The present result showed the induction of TNF-α mRNA expression in the kidney of transgenic mouse.

Journal

• The Japanese Journal of Pharmacology

  The Japanese Journal of Pharmacology 76 (1), 97-99, 1998

  The Japanese Pharmacological Society

Keywords

• Transgenic mouse
• Interferon
• Tumor necrosis factor

Details

• CRID: 1390001204285264896
• NII Article ID: 10008191236
• NII Book ID: AA00691188
• DOI: 10.1254/jjp.76.97
• COI: 1:CAS:528:DyaK1cXmsFWjtQ%3D%3D
• ISSN: 13473506
• ISSN: 00215198
• NDL BIB ID: 4395862
• PubMed: 9517409
• Web Site: https://ndlsearch.ndl.go.jp/books/R000000004-I4395862
• Web Site: https://api.elsevier.com/content/article/PII:S002151981931251X?httpAccept=text/xml
• Web Site: https://api.elsevier.com/content/article/PII:S002151981931251X?httpAccept=text/plain
• Web Site: https://www.jstage.jst.go.jp/article/jjp/76/1/76_1_97/_pdf
• Web Site: https://search.jamas.or.jp/link/ui/1998112011
• Text Lang: en
• Data Source:
  • JaLC
  • NDL
  • Crossref
  • PubMed
  • CiNii Articles
• Abstract License Flag: Disallowed