Renal Tubular Acidosis type II Secondary to .LAMBDA.-Light Chain Excretion in an Elderly Patient with Multiple Myeloma.

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  • Doi Kent
    Department of Geriatrics, Faculty of Medicine, Univerity of Tokyo
  • Teramoto Shinji
    Department of Geriatrics, Faculty of Medicine, Univerity of Tokyo
  • Hosoi Takayuki
    Department of Geriatrics, Faculty of Medicine, Univerity of Tokyo
  • Miyao Mariko
    Department of Geriatrics, Faculty of Medicine, Univerity of Tokyo
  • Matsuse Takeshi
    Department of Geriatrics, Faculty of Medicine, Univerity of Tokyo
  • Toba Kenji
    Department of Geriatrics, Faculty of Medicine, Univerity of Tokyo
  • Ouchi Yasuyoshi
    Department of Geriatrics, Faculty of Medicine, Univerity of Tokyo

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  • 近位尿細管障害を伴った老年者多発性骨髄腫の１例

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Abstract

A 73-year-old women was admitted to the geriatric ward of the University of Tokyo Hospital with anemia, osteepeina, and renal dysfunction. Although symptoms typical of multiple myeloma such as punched-out lesions and hyperproteinemia were not found, protein electrophoresis revealed that λtype Bence-Jones protein was excreted in urine. Multiple myeloma was diagnosed. Furthermore, renal dysfunction was accompanied renal tubular acidosis type II (proximal type). Renal dysfunction in patient with multiple myeloma in usually caused by so-called myeloma casts in the distal tubules, but renal tubular acidosis type II is rarely observed. It is possible that injury of the proximal renal tubular eithelium by Bence-Jones protein resulted in renal tubular acidosis type II in this patient.

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