(-)-epigallocatechin-3-gallate potentiates the cytotoxicity induced benzyl isothiocyanate and hydrogen peroxide in human Jurkat T lymphocytes

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  • WU Haitao
    Graduate School of Natural Science and Technology, Okayama University College of Bio and Food Technology, Dalian Polytechnic University
  • YOKOYAMA Tomoko
    Graduate School of Natural Science and Technology, Okayama University
  • ZHU Beiwei
    College of Bio and Food Technology, Dalian Polytechnic University
  • SHIMOISHI Yasuaki
    Graduate School of Natural Science and Technology, Okayama University
  • MURATA Yoshiyuki
    Graduate School of Natural Science and Technology, Okayama University
  • NAKAMURA Yoshimasa
    Graduate School of Natural Science and Technology, Okayama University

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タイトル別名
  • (-)-Epigallocatechin-3-gallate Potentiates the Cytotoxicity Induced by Benzyl Isothiocyanate and Hydrogen Peroxide in Human Jurkat T Lymphocytes
  • (−)-Epigallocatechin-3-gallate Potentiates the Cytotoxicity Induced by Benzyl Isothiocyanate and Hydrogen Peroxide in Human Jurkat T Lymphocytes

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(−)-Epigallocatechin-3-gallate (EGCG)-induced apoptosis was along both the extracellular signal-regulated protein kinase (ERK) and c-jun N-terminal kinase (JNK) pathways in Jurkat cells. Co-treatment with EGCG potentiated the cytotoxicity induced by benzyl isothiocyanate (BITC) and H2O2, both being inhibited by ERK and JNK inhibitors. These results suggest the significant role of mitogen-activated protein kinase (MAPK) signaling in the apoptosis induction regulated by EGCG alone and in combination.

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