A long-term follow-up study of acute myocarditis: an electrocardiographic and echocardiographic study

• HAYAKAWA M.

  First Division, Department of Internal Medicine, Kobe University School of Medicine

• INOH TSUTOMU

  Department of Internal Medicine, Miki City Hospital

• YOKOTA YOSHIYUKI

  First Division, Department of Internal Medicine, Kobe University School of Medicine

• KAWANISHI HIDEO

  Department of Internal Medicine, Miki City Hospital

• KUMAKI TOMOYUKI

  First Division, Department of Internal Medicine, Kobe University School of Medicine

• TAKARADA AKIRA

  First Division, Department of Internal Medicine, Kobe University School of Medicine

• SEO TOSHIHIKO

  First Division, Department of Internal Medicine, Kobe University School of Medicine

• FUKUZAKI HISASHI

  First Division, Department of Internal Medicine, Kobe University School of Medicine

抄録

In an attempt to prove that myocarditis may be a cause of idiopathic cardiomyopathy, 10 cases with acute myocarditis were involved in a long-term follow-up study. there were 9 males and 1 female patient, ranging in age from 22 to 63 years. The etiology of myocarditis was idiopathic in 9 cases and rubellavirus in case. clinical findings in the acute stage consisted of congestive heart failure in 6 case, Adams-Stokes syndrome in 2 cases and cardiomegaly in 2 cases. The mean follow-up period was 55 months. Follow-up studies included physical examinations, 12-lead ECG, chest X-rays and two-dimensional echocardiograms. Various patterns of residual ECG abnormalities in the chronic stage were found, such as conduction disturbance in 1 case, pseudoinfarction patter in 4 cases, ST-T changes in 2 cases and premature ventricular contractions in 2 cases. The cardiothoracic ratio of all cases was 60±4% in the acute stage. Two cases (Case1 and 2) died 16 and 36 months after the acute onset, respectively. Four cases had residual cardiomegaly even in the last study period. An echocardiographic follow-up study of 7 cases disclosed progressive left ventricular (LV) dilation and dysfunction in 3 cases, regression of LV dilatation in 2 cases and stable LV function in 2 cases. Two cases out of 3 with progressive LV dilatation and dysfunction expired after the acute illness. It was therefore suggested that acute myocarditis may cause LV dilatation and/or wall hypertrophy and that idiopathic cardiomyopathy may represent the end-stages of previous myocarditis.

収録刊行物

• Jpn Circ J

  Jpn Circ J 48 1362-1367, 1984

  社団法人日本循環器学会

キーワード

• Actute myocarditis
• Fllow-up study
• Echocardiography
• Idiopathic cardiomyopathy

詳細情報

• CRID: 1570854177119580800
• NII論文ID: 110002578197
• NII書誌ID: AA00690731
• ISSN: 00471828
• Web Site: https://search.jamas.or.jp/link/ui/1985172170
• 本文言語コード: en
• データソース種別:
  • CiNii Articles