51.ブラシノステロイド情報伝達突然変異体bil4の原因遺伝子の機能解析(口頭発表)

DOI

書誌事項

タイトル別名
  • 51. Brassinosteroid signaling mutants bil4

抄録

Brassinosteroids (BRs) are perceived by a cell surface receptor kinase BRI1, and then various components are predicted to be involved to transduce the BR signal in the cell. To identify additional components involved in BRs signal, we had analyzed various dwarf and semidwarf mutants from Arabidopsis mutant, which is insensitive to the BRs synthetic inhibitor Brz. We had identified Brz insensitive mutant bill from EMS-mutation lines, and bil5 from fast neutron-mutation lines. We tried to screen new bil mutants from Arabidopsis activation-tag lines and selected bil4 that showed Brz-insensitive phenotype in the dark. Genetic analysis suggested that the candidate genes of bil4 mutation are a novel protein. Transgenic plants overexpressing the bil4 candidte gene display a slender dwarf phenotype and are insensitive to Brz. From genetic and functional analyses to bil4 mutant, we would like to clarify in detail the mechanism of brassinosteroid signaling transduction.

収録刊行物

詳細情報 詳細情報について

  • CRID
    1390001204423152000
  • NII論文ID
    110007468311
  • DOI
    10.18978/jscrpanb.44.0_65
  • ISSN
    21896313
    09191887
  • 本文言語コード
    ja
  • データソース種別
    • JaLC
    • CiNii Articles
  • 抄録ライセンスフラグ
    使用不可

問題の指摘

ページトップへ