Chemically induced cell proliferation : implications for risk assessment : proceedings of the Chemically Induced Cell Proliferation Conference, held in Austin, Texas, November 29-December 2, 1989

著者

書誌事項

Chemically induced cell proliferation : implications for risk assessment : proceedings of the Chemically Induced Cell Proliferation Conference, held in Austin, Texas, November 29-December 2, 1989

editors, Byron E. Butterworth ... [et al.]

（Progress in clinical and biological research, v. 369）

Wiley-Liss, c1991

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注記

Includes bibliographical references and index

内容説明・目次

内容説明

Containing a compilation of articles written by leaders in their respective fields, this book provides current information on various roles that chemically-induced cell proliferation might play in the carcinogenic process. With today's intense economic competition, limited environmental resources and conflicting advice to the public on personal dangers, cancer policy needs to be based on the best available information. The association of chemically induced cell proliferation with carcinogenic activity is so prevalent that it must be considered in evaluating the carcinogenic danger in chemicals to which people may be exposed. The overviews in this book offer insights into these mechanisms that should yield new predictive assays, improved design and interpretation for cancer bioassays and more realistic risk evaluations.

目次

  • Partial table of contents:
  • CELL PROLIFERATION IN HUMAN CARCINOGENESIS
  • The Role of Chemically Induced Epithelial Hyperplasia in the Development of Human Cancer
  • BASIC MECHANISMS OF CELL PROLIFERATION: PART I. Cellular Proliferation and Stomach Carcinogenesis Induced by Antioxidants
  • BASIC MECHANISMS OF CELL PROLIFERATION: PART II
  • Liver Tumor Promoter Phenobarbital: A Biphasic Modulator of Hepatocyte Proliferation
  • REGENERATION, PROGRAMMED CELL DEATH AND SPECIFIC METHODOLOGY
  • Chemically Induced Cell Proliferation as a Criterion in Selecting Doses for Long-Term Bioassays
  • SPECIFIC EXAMPLES: PART I. Chemically Induced Cell Proliferation in Upper Respiratory Tract Carcinogenesis
  • SPECIFIC EXAMPLES: PART II
  • Role of Iron in Cell Proliferation and Tumorigenesis
  • RISK ASSESSMENT
  • Chemicals, Cell Proliferation, Risk Estimation, and Multistage Carcinogenesis.

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