New frontiers in stress research : modulation of brain function
著者
書誌事項
New frontiers in stress research : modulation of brain function
Harwood Academic, 1998
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注記
Includes index
内容説明・目次
内容説明
The stress response is an adaptive, complex, and essential process that serves to prepare an organism for the variety of dangers it may encounter. However, this same process may also be detrimental. The challenge of identifying the circumstances which alter the advantageous stress response to a damaging process has been taken up by scientists from various disciplines.
The interdisciplinary focus on the modifications of brain functions as the medium for the immediate, as well as the long-term, effects of stress, enables a simultaneous analysis of the behavioral, histological, hormonal, and immunological processes involved. The dichotomy of the stress response is elucidated through the interactions of various neuronal sites, and the neurotransmitter and hormonal regulation of the central nervous system. Research on the interaction between stress and neuroimmunological processes is included and the implications of these stress effects to human conditions such as Post Traumatic Stress Disorder (PTSD), Alzheimer's disease, and memory impairment are addressed.
目次
Section I: Neuroendocrinology of the Stress Response 1. The Role and Regulation of Monoamines in Stress 2. Glucocorticoids, Serotonin and Their Interactions in the Hippocampus Section II: Stress-Hormone Action: Basic Mechanisms 3. Corticosteroids and Calcium Homeostasis: Implication for Neuroprotection and Neurodegeneration 4. Deleterious Consequences of Corticosteroid Exposure: Possible 5. Therapeutic Protection Section III: Stress and Development 6. The Developmental Neurobiology of the Response to Stress 7. Long-Term Effects of Gestational Stress on Behaviour and Pituitary-Adrenal Function 8. Response 9. Immunomodulation of Catecholamines and Corticosteroids 10. Steroids, Stress and the Immune Mechanisms of Stress-Induced Section IV: Neuroimmunology of the Stress 11. Stress, Natural Killer Cell Activity, and Tumor Metastasis: The Role
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