Disease markers in exhaled breath : basic mechanisms and clinical applications
著者
書誌事項
Disease markers in exhaled breath : basic mechanisms and clinical applications
(NATO science series, series 1 . Life and behavioural sciences ; v. 346)
IOS Press, c2002
- : IOS Press
- : Ohmsha
大学図書館所蔵 全2件
  青森
  岩手
  宮城
  秋田
  山形
  福島
  茨城
  栃木
  群馬
  埼玉
  千葉
  東京
  神奈川
  新潟
  富山
  石川
  福井
  山梨
  長野
  岐阜
  静岡
  愛知
  三重
  滋賀
  京都
  大阪
  兵庫
  奈良
  和歌山
  鳥取
  島根
  岡山
  広島
  山口
  徳島
  香川
  愛媛
  高知
  福岡
  佐賀
  長崎
  熊本
  大分
  宮崎
  鹿児島
  沖縄
  韓国
  中国
  タイ
  イギリス
  ドイツ
  スイス
  フランス
  ベルギー
  オランダ
  スウェーデン
  ノルウェー
  アメリカ
注記
Includes bibliographies and index
内容説明・目次
内容説明
This monograph contains the contributions of invited speakers and participants at the NATO Advanced Study Institute on Disease Markers in Exhaled Breath: Basic Mechanisms and Clinical Applications, held in Greece in 2001. This ASI was designed to summarize and disseminate expert knowledge regarding this rapidly evolving field of lung biology. Breath testing dates from the earliest history of medicine and puzzled brilliant scientific minds including Linus Pauling. The recent developments hold enormous promise that analysis of exhaled breath could open a valuable new window onto human metabolism and illuminate its functions in health and disease. Accordingly, analysis of NO, CO and VOCs in exhaled breath has become a primary focus of respiratory research and an essential aspect of investigations into many systemic diseases both in the laboratory and in the clinics. These activities are reflected in the rapid expansion of presentations at international meetings, scientific and clinical publications, editorials and recommendations by major respiratory societies.
Topics included: acute and chronic lung inflammation with major emphasis on asthma and chronic obstructive pulmonary disease, acute lung injury such as occurring during thoracic organ transplantation and related end stage lung disease, mechanisms of acute and chronic rejection, ischaemia-reperfusion injury and systemic inflammation.
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