Current neurochemical and pharmacological aspects of biogenic amines : their function, oxidative deamination, and inhibition
Author(s)
Bibliographic Information
Current neurochemical and pharmacological aspects of biogenic amines : their function, oxidative deamination, and inhibition
（Progress in brain research, v. 106）
Elsevier, 1995
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National Institutes of Natural Sciences Okazaki Library and Information Center図
491.371/Pr/1069207822021
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Note
Includes bibliographical references and index
Description and Table of Contents
Description
This book contains anonymous-peer-reviewed invited contributions from the 6th Amine Oxidase Workshop and the 5th Trace Amine Conference. New developments in both basic and applied studies related to monoamine oxidases (MAO), semicarbazide-sensitive amine oxidase (SSAO) and the trace amines are addressed.
Table of Contents
- The colourful past and bright future of monoamine oxidase research
- radical thoughts about the life of MAO
- redox properties of flavin cofactor of monoamine oxidases A and B and their relationship to the kinetic mechanism
- stereochemistry and cofactor identity status of semicarbazide-sensitive amine oxidases
- expression of human monoamine oxidase (MAO) A gene controlled by transcription factor Sp1.
- the promoter of the human monoamine oxidase A gene
- analysis of MAO-A mutations in humans
- the correlation between platelet MAO activity and personality: short review of findings and a discussion on possible mechanism
- platelet MAO activities and MAO-B protein concentrations in Parkinson's disease and controls
- aromatic L-amino acid decarboxylase modulation and Parkinson's disease
- some new mechanism underlying the actions of (-)-deprenyl: possible relevance to neurogeneration
- neurochemical, neuroprotective and neurorescue effects of aliphatic N-methylpropargylamine: new MAO-B inhibitors without amphetamine-like properties
- enentioselective recognition of two anticonvulsants, FCE 26743 and FCE 28073, by MAO, and relationship between MAO-B inhibition and FCE 26743 concentrations in rat brain
- Selectivity of MDL-72,974A for MAO-B inhibition based on substrate and metabolite concentrations in plasma
- the distribution of orally administered (-)-deprenyl-propenyl-14C and (-)deprenyl-phenyl-3H in rat brain
- novel sites of action for deprenyl in MPTP-Parkinsonism: metabolite mediated protection against striatal neurotoxicity and suppression of MPTP-induced increase of dopamine turnover in C5BL mice
- effects of transient global ischemia and a monoamine oxidase inhibitor ifenprodil on rat brain monoamine metabolism
- effects of MAO inhibitor phenelzine on glutamine and GABA concentrations in rat brain
- metabolism of agmatine (clonidine-displacing substance) by diamine oxidase and the possible implications for studies of imidazoline receptors
- increase of survival of dopaminergic neuroblastoma in co-cultures with C-6 glioma by R(-)-deprenyl
- canine pituitary-dependent hyperadrenocorticism: a spontaneous animal model for neurodegenerative disorders and their treatment with l-deprenyl
- canine cognitive dysfunction as a model for human age-related cognitive decline, dementia and Alzheimer's disease: clinical presentation, cognitive testing, pathology and response to l-deprenyl therapy
- dopamine-derived 6,7-dihydroxy-1,2,3,4-tetrahydroisoquinolines, oxidation and neurotoxicity
- monoamines, cytoskeletal elements and psychiatric disorders, a neurochemical fugue. (Part contents).
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